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2 edition of Pathogenicity mutants of fulvia fulva (Cooke) Cif. (syn. Cladosporium fulvum Cooke). found in the catalog.

Pathogenicity mutants of fulvia fulva (Cooke) Cif. (syn. Cladosporium fulvum Cooke).

Lawrence Kenyon

Pathogenicity mutants of fulvia fulva (Cooke) Cif. (syn. Cladosporium fulvum Cooke).

by Lawrence Kenyon

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Published by University of East Anglia in Norwich .
Written in English


Edition Notes

Thesis (Ph.D), University of East Anglia, School of Biological Sciences, 1990.

ID Numbers
Open LibraryOL14593072M

Politics, Philosophy, Language and Communication Studies. Graduate School. Medicine and Health Sciences.   Iron acquisition is an important pathogenicity factor in the bacterial and fungal pathogenesis of animals. It was surprising that mutants of Ustilago maydis, which lacked a siderophore gene, retained their pathogenicity (Mei et al., ). Clearly the fungus must have other means of acquiring iron that still need to be elucidated. CONCLUSIONS.

Show simple item record. Mannitol 1-phosphate metabolism is required for sporulation in planta of the wheat pathogen Stagonospora nodorum. INTRODUCTION. Cladosporium fulvum [syn. Passalora fulva (Braun et al., )] is the causal organism of tomato leaf mould, a fungal disease first described by Cooke ().Generally, foliage is the only tissue affected by the fungus, although occasionally also stems, blossoms, petioles and fruit are attacked (Butler and Jones, ; Jones et al., ).

Mutants ofFusarium oxysporum f. rsici were obtained by UV irradiation. The mutants of race 1 and race 2 caused disease symptoms on plants with resistance genes against the corresponding wild type strains. Mutants of race 1 of the pathogen were stable, whereas mutants of race 2 lost the ability to cause disease symptoms in plants carrying the 1–2 resistance gene, after prolonged. The fungus Mycosphaerella fijiensis causes black Sigatoka (BS) disease, a major pathogen in the banana industry worldwide. Numerous molecular and biochemical studies have been done for the M. fijiensis, Musa acuminata interaction, but this is the first study describing the zymographic behavior of β-1,3-glucanase, chitinase and protease in the apoplast and symplast of healthy, BS-infected but.


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RACER # 3056393

RACER # 3056393

Pathogenicity mutants of fulvia fulva (Cooke) Cif. (syn. Cladosporium fulvum Cooke) by Lawrence Kenyon Download PDF EPUB FB2

A range of mutants with impaired ability to infect tomato cotyledons and leaves was produced by exposing conidia of Fulvia fulva (race 4) to either u.v. light or 1,3-butadiene diepoxide or N-methyl-N-nitro-N-nitrosoguanidine. One spontaneous mutant was also by: 7.

Buy Pathogenicity mutants of Fulvia fulva (Cooke) Cif: syn. Cladosporum fulvum Cooke by Lawrence Kenyon (ISBN:) from Amazon's Book Store.

Everyday low prices and free delivery on eligible : Lawrence Kenyon. A range of mutants with impaired ability to infect tomato cotyledons and leaves was produced by exposing conidia of Fulvia fulva (race 4) to either u.v.

light or 1,3-butadiene diepoxide or N-methyl-N-nitro-N-nitrosoguanidine. One spontaneous mutant was also recovered. In axenic culture, growth rates and sporulation of the mutants were similar to the wild-type race 4.

Pathogenicity mutants of the tomato leaf mould fungus Fulvia fulva (Cooke) Ciferri (syn. Cladosporium fulvum Cooke) By L Kenyon, BG Lewis, A Coddington, R Harling and JG Turner. Cite. BibTex; Full citation; Abstract.

A range of mutants with impaired ability to infect tomato cotyledons and leaves was produced by exposing conidia of Fulvia fulva. Pathogenicity mutants of Fulvia fulva (Cooke) Cif.: (syn. Cladosporum fulvum Cooke). Author: Kenyon, Lawrence. ISNI: Awarding Body: University of East Anglia Current Institution: University of East Anglia Date of Award.

Pathogenicity mutants of Fulvia fulva (Cooke) Cif.: (syn. Cladosporum fulvum Cooke). Thesis (Thesis) Find all citations by this author (default). Deletion ofHCf-1, a hydrophobin gene ofCladosporium fulvum, does not affect pathogenicity on tomato HCfless mutants, mycelium and cell walls ofCladosporium fulvumFulvia fulva.

Physiological Plant Pathology, 18 (), pp. A range of mutants with impaired ability to infect tomato cotyledons and leaves was produced by exposing conidia of Fulvia fulva (race 4) to either u.v. light or 1,3-butadiene diepoxide or N. Both disruption mutants were still pathogenic on tomato seedlings, indicating that the C.

fulvum ecp2 gene is not essential for pathogenicity in tomato. During the colonization of tomato leaves, the fungal pathogen Cladosporium fulvum excretes low-molecular-weight proteins in the intercellular spaces of the. Fulvia fulva (Cooke) Cif., Atti dell'Istituto Botanico della Università e Laboratorio Crittogamico di Pavia 10 (2): () [MB#].

Genetics and Plant Pathogenesis by G. Jellis,available at Book Depository with free delivery worldwide. We use cookies to give you the best possible experience. By using our website you agree to our use of cookies. Home. Pathogenicity mutants of the tomato leaf mould fungus Fulvia fulva (Cooke, Ciferri) syn.

Cladosporium fulvum (Cooke). Date Author. Kenyon L. Lewis BG. Harling R. Coddington A. Turner J. Metadata Show full item record. Journal Title/Title of Proceedings. Physiological and Molecular Plant Pathology.

Abstract. Cladosporium fulvum Cke (syn. Fulvia fulva) is a fungal pathogen of the tomato, Lycopersicon esculentum. causing the disease leaf mould.A variety of natural features of the interaction together with techniques and knowledge built up over past few years have established these organisms as a near-ideal systems for the study of pathogenicity, host specificity and race cultivar specificity.

A description of C. fulvum [Fulvia fulva], a potentially serious fungal pathogen of tomato, which has attracted attention as a model system in fungal pathogenicity, is presented.

The disease history, taxonomy and phylogenetics of this species are given. The mode of infection, mechanism of resistance and pathogenicity are discussed.

Passalora fulva. Leaf mold caused by Passalora fulva (previously Fulvia fulva) is primarily a disease of greenhouse-grown tomatoes and is most serious in plastic greenhouses where the relative humidity is pathogen is found worldwide and is occasionally a problem on field grown tomatoes.

Identification: Foliage is usually the only tissue affected. This is the full account of studies on the physiologic races of Cladosporium fulvum[Fulvia fulva] in south-western Ontario, a preliminary abstract of which has already been noticed [R.A.M., 28, p. So far seven races differing in their host reactions have been isolated.

Vetomold tomato is susceptible to races 5, 6, 7, and 8; V to 6, 7, and 9; Stirling Castle to 2, 6, and 8; V transliterated in Latin, and the titles of papers or books are translated into English. The letters in transliteration are replaced according to the Law on Transliteration.

The original language of publications, translated into English, is given after the references in brackets. Requirements for Contributors to the Journal of Mountain Agriculture on the Balkans (JMAB) Journal of Mountain Agriculture on the Balkans publishes papers which are classified into the.

In tomato (Lycopersicon esculentum) several acidic and basic apoplastic pathogenesis-related (PR) proteins are induced upon inoculation with virulent or avirulent races of Cladosporium fulvum (Cooke) (syn.

Fulvia fulva [Cooke] Cif). One of the most predominant and best characterized tomato PR proteins is P14, a basic protein that shows homology to the tobacco (Nicotiana tabacum) PR Nick Talbot Talbot, Nicholas J.

Talbot, Nick. Talbot, Nick (Nicholas J.) VIAF ID: (Personal) Permalink:. Enter search terms. Keep search filters New search. Advanced search.Joosten MH, De Wit PJ.

Identification of Several Pathogenesis-Related Proteins in Tomato Leaves Inoculated with Cladosporium fulvum (syn. Fulvia fulva) as 1,3-beta-Glucanases and Chitinases. Plant Physiol. Mar; 89 (3)– [PMC free article] Kunkel .Inoculation of tomato (Lycopersicon esculentum) leaves with Cladosporium fulvum (Cooke) (syn.

Fulvia fulva [Cooke] Cif) results in a marked accumulation of several pathogenesis-related (PR) proteins in the apoplast. Two predominant PR proteins were purified from apoplastic fluid by ion exchange chromatography followed by chromatofocusing.

One protein (molecular mass [ M r] 35 kilodaltons [kD.